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    Please use this identifier to cite or link to this item: http://ir.nhri.org.tw/handle/3990099045/17020


    Title: Crosstalk between FTH1 and PYCR1 dysregulates proline metabolism and mediates cell growth in KRAS-mutant pancreatic cancer cells[Erratum:Experimental and Molecular Medicine. 2024 Sep 18;56:2065-2081.]
    Other Titles: Author Correction: Crosstalk between FTH1 and PYCR1 dysregulates proline metabolism and mediates cell growth in KRAS-mutant pancreatic cancer cells
    Authors: Park, JM;Su, YH;Fan, CS;Chen, HH;Qiu, YK;Chen, LL;Chen, HA;Ramasamy, TS;Chang, JS;Huang, SY;Chang, WW;Lee, AY;Huang, TS;Kuo, CC;Chiu, CF
    Contributors: Institute of Cellular and Systems Medicine;National Institute of Cancer Research
    Abstract: After online publication of this article, the authors noticed an error in the results section and supplementary information. The correct statement of this article should have read as below. Modification of language around FTH1 and PYCR1 interaction in the paragraph of Result, under the title of “FTH1-PYCR1 crosstalk mediates pancreatic cancer progression” on pages 2071-2073. We would like to clarify of experimental findings supporting the feedback mechanism between FTH1 and PYCR1.
    Date: 2025-03-27
    Relation: Experimental and Molecular Medicine. 2025 Mar 27;57(3):700-701.
    Link to: http://dx.doi.org/10.1038/s12276-025-01437-w
    JIF/Ranking 2023: http://gateway.webofknowledge.com/gateway/Gateway.cgi?GWVersion=2&SrcAuth=NHRI&SrcApp=NHRI_IR&KeyISSN=1226-3613&DestApp=IC2JCR
    Appears in Collections:[郭呈欽] 期刊論文
    [黃智興] 期刊論文
    [李岳倫] 期刊論文
    [張文祥] 期刊論文

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