國家衛生研究院 NHRI:Item 3990099045/6729
English  |  正體中文  |  简体中文  |  全文笔数/总笔数 : 12145/12927 (94%)
造访人次 : 858839      在线人数 : 817
RC Version 6.0 © Powered By DSPACE, MIT. Enhanced by NTU Library IR team.
搜寻范围 查询小技巧:
  • 您可在西文检索词汇前后加上"双引号",以获取较精准的检索结果
  • 若欲以作者姓名搜寻,建议至进阶搜寻限定作者字段,可获得较完整数据
  • 进阶搜寻
    主页登入上传说明关于NHRI管理 到手机版


    jsp.display-item.identifier=請使用永久網址來引用或連結此文件: http://ir.nhri.org.tw/handle/3990099045/6729


    题名: Effects of dipeptidyl peptidase 4 inhibition with MK-0431 on syngeneic mouse islet transplantation
    作者: Juang, JH;Kuo, CH;Liu, YH;Chen, CT
    贡献者: Institute of Biotechnology and Pharmaceutical Research
    摘要: Dipeptidyl peptidase-4 (DPP-4) inhibitors increase circulating levels of active incretin hormones-glucagon-like peptide-1 (GLP-1) and glucose-dependent insulinotropic polypeptide (GIP) by blocking their degradation. In addition to their insulinotropic actions, GLP-1 and GIP also promote beta-cell proliferation and survival. The aim of this study was to test if DPP-4 inhibition with MK-0431 is beneficial for diabetic recipients with a marginal number of fresh islets.
    日期: 2011-09
    關聯: Diabetologia. 2011 Sep;54(Suppl. 1):S181.
    Link to: http://dx.doi.org/10.1007/s00125-011-2276-4
    JIF/Ranking 2023: http://gateway.webofknowledge.com/gateway/Gateway.cgi?GWVersion=2&SrcAuth=NHRI&SrcApp=NHRI_IR&KeyISSN=0012-186X&DestApp=IC2JCR
    Cited Times(WOS): https://www.webofscience.com/wos/woscc/full-record/WOS:000307671301084
    显示于类别:[陳炯東] 會議論文/會議摘要

    文件中的档案:

    档案 描述 大小格式浏览次数
    ISI000307671301084.pdf932KbAdobe PDF560检视/开启


    在NHRI中所有的数据项都受到原著作权保护.

    TAIR相关文章

    DSpace Software Copyright © 2002-2004  MIT &  Hewlett-Packard  /   Enhanced by   NTU Library IR team Copyright ©   - 回馈